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mCh-Rab7A Citations (7)

Plasmid Article: ER Contact Sites Define the Position and Timing of Endosome Fission.
Rowland AA, Chitwood PJ, Phillips MJ, Voeltz GK Cell. 2014 Nov 20;159(5):1027-41. doi: 10.1016/j.cell.2014.10.023.
PubMed Journal

Articles Citing mCh-Rab7A

Articles
VMP1 Establishes ER-Microdomains that Regulate Membrane Contact Sites and Autophagy. Tabara LC, Escalante R. PLoS One. 2016 Nov 18;11(11):e0166499. doi: 10.1371/journal.pone.0166499. eCollection 2016. PubMed
Impaired JIP3-dependent axonal lysosome transport promotes amyloid plaque pathology. Gowrishankar S, Wu Y, Ferguson SM. J Cell Biol. 2017 Oct 2;216(10):3291-3305. doi: 10.1083/jcb.201612148. Epub 2017 Aug 7. PubMed
Mitochondria-lysosome contacts regulate mitochondrial fission via RAB7 GTP hydrolysis. Wong YC, Ysselstein D, Krainc D. Nature. 2018 Feb 15;554(7692):382-386. doi: 10.1038/nature25486. Epub 2018 Jan 24. PubMed
Vacuole membrane protein 1 marks endoplasmic reticulum subdomains enriched in phospholipid synthesizing enzymes and is required for phosphoinositide distribution. Tabara LC, Vicente JJ, Biazik J, Eskelinen EL, Vincent O, Escalante R. Traffic. 2018 Aug;19(8):624-638. doi: 10.1111/tra.12581. Epub 2018 Jun 10. PubMed
VPS13A and VPS13C are lipid transport proteins differentially localized at ER contact sites. Kumar N, Leonzino M, Hancock-Cerutti W, Horenkamp FA, Li P, Lees JA, Wheeler H, Reinisch KM, De Camilli P. J Cell Biol. 2018 Oct 1;217(10):3625-3639. doi: 10.1083/jcb.201807019. Epub 2018 Aug 9. PubMed

Associated Plasmids

Chemical genetic identification of GAK substrates reveals its role in regulating Na(+)/K(+)-ATPase. Lin AW, Gill KK, Castaneda MS, Matucci I, Eder N, Claxton S, Flynn H, Snijders AP, George R, Ultanir SK. Life Sci Alliance. 2018 Dec 31;1(6):e201800118. doi: 10.26508/lsa.201800118. eCollection 2018 Dec. PubMed
VPS13A is closely associated with mitochondria and is required for efficient lysosomal degradation. Munoz-Braceras S, Tornero-Ecija AR, Vincent O, Escalante R. Dis Model Mech. 2019 Feb 22;12(2). pii: dmm.036681. doi: 10.1242/dmm.036681. PubMed

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