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pRK5-EGFP-Tau AP Citations (4)

Originally described in: Tau mislocalization to dendritic spines mediates synaptic dysfunction independently of neurodegeneration.
Hoover BR, Reed MN, Su J, Penrod RD, Kotilinek LA, Grant MK, Pitstick R, Carlson GA, Lanier LM, Yuan LL, Ashe KH, Liao D Neuron. 2010 Dec 22;68(6):1067-81. doi: 10.1016/j.neuron.2010.11.030.
PubMed Journal

Articles Citing pRK5-EGFP-Tau AP

12/15-Lipoxygenase Inhibition Reverses Cognitive Impairment, Brain Amyloidosis, and Tau Pathology by Stimulating Autophagy in Aged Triple Transgenic Mice. Di Meco A, Li JG, Blass BE, Abou-Gharbia M, Lauretti E, Pratico D. Biol Psychiatry. 2017 Jan 15;81(2):92-100. doi: 10.1016/j.biopsych.2016.05.023. Epub 2016 Jun 4. PubMed
Hyperphosphorylated tau causes reduced hippocampal CA1 excitability by relocating the axon initial segment. Hatch RJ, Wei Y, Xia D, Gotz J. Acta Neuropathol. 2017 May;133(5):717-730. doi: 10.1007/s00401-017-1674-1. Epub 2017 Jan 16. PubMed
Tau hyperphosphorylation induces oligomeric insulin accumulation and insulin resistance in neurons. Rodriguez-Rodriguez P, Sandebring-Matton A, Merino-Serrais P, Parrado-Fernandez C, Rabano A, Winblad B, Avila J, Ferrer I, Cedazo-Minguez A. Brain. 2017 Dec 1;140(12):3269-3285. doi: 10.1093/brain/awx256. PubMed
Long-term mitochondrial stress induces early steps of Tau aggregation by increasing reactive oxygen species levels and affecting cellular proteostasis. Samluk L, Ostapczuk P, Dziembowska M. Mol Biol Cell. 2022 Jul 1;33(8):ar67. doi: 10.1091/mbc.E21-11-0553. Epub 2022 Apr 21. PubMed

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