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HBV 1.3-mer P-null replicon Citations (4)

Originally described in: DDX3 DEAD-Box RNA helicase inhibits hepatitis B virus reverse transcription by incorporation into nucleocapsids.
Wang H, Kim S, Ryu WS J Virol. 2009 Jun;83(11):5815-24. doi: 10.1128/JVI.00011-09. Epub 2009 Mar 18.
PubMed Journal

Articles Citing HBV 1.3-mer P-null replicon

Tripartite motif protein 52 (TRIM52) promoted fibrosis in LX-2 cells through PPM1A-mediated Smad2/3 pathway. Zhou J, Lan Q, Li W, Yang L, You J, Zhang YM, Ni W. Cell Biol Int. 2019 Jul 22. doi: 10.1002/cbin.11206. PubMed
HBV-Pol is crucial for HBV-mediated inhibition of inflammasome activation and IL-1beta production. Lei Q, Li T, Kong L, Li L, Ding X, Wang X, Zhang X, Qin B. Liver Int. 2019 Dec;39(12):2273-2284. doi: 10.1111/liv.14214. Epub 2019 Aug 30. PubMed
ROS/NF-kappaB Signaling Pathway-Mediated Transcriptional Activation of TRIM37 Promotes HBV-Associated Hepatic Fibrosis. Xie H, Xie D, Zhang J, Jin W, Li Y, Yao J, Pan Z, Xie D. Mol Ther Nucleic Acids. 2020 Aug 19;22:114-123. doi: 10.1016/j.omtn.2020.08.014. PubMed
Hepatitis B virus X protein recruits methyltransferases to affect cotranscriptional N6-methyladenosine modification of viral/host RNAs. Kim GW, Siddiqui A. Proc Natl Acad Sci U S A. 2021 Jan 19;118(3). pii: 2019455118. doi: 10.1073/pnas.2019455118. PubMed

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