EYFP-hCaM Citations (5)

Originally described in: The tumor-sensitive calmodulin-like protein is a specific light chain of human unconventional myosin X.
Rogers MS, Strehler EE J Biol Chem. 2001 Apr 13;276(15):12182-9. Epub 2001 Jan 22. PubMed Journal

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Articles Citing EYFP-hCaM

Articles
Reduced axonal surface expression and phosphoinositide sensitivity in Kv7 channels disrupts their function to inhibit neuronal excitability in Kcnq2 epileptic encephalopathy. Kim EC, Zhang J, Pang W, Wang S, Lee KY, Cavaretta JP, Walters J, Procko E, Tsai NP, Chung HJ. Neurobiol Dis. 2018 Oct;118:76-93. doi: 10.1016/j.nbd.2018.07.004. Epub 2018 Jul 6. PubMed
Gain-of-Function Mutations in KCNN3 Encoding the Small-Conductance Ca(2+)-Activated K(+) Channel SK3 Cause Zimmermann-Laband Syndrome. Bauer CK, Schneeberger PE, Kortum F, Altmuller J, Santos-Simarro F, Baker L, Keller-Ramey J, White SM, Campeau PM, Gripp KW, Kutsche K. Am J Hum Genet. 2019 Jun 6;104(6):1139-1157. doi: 10.1016/j.ajhg.2019.04.012. Epub 2019 May 30. PubMed
Flexible linkers in CaMKII control the balance between activating and inhibitory autophosphorylation. Bhattacharyya M, Lee YK, Muratcioglu S, Qiu B, Nyayapati P, Schulman H, Groves JT, Kuriyan J. Elife. 2020 Mar 9;9. pii: 53670. doi: 10.7554/eLife.53670. PubMed
Identifying mutation hotspots reveals pathogenetic mechanisms of KCNQ2 epileptic encephalopathy. Zhang J, Kim EC, Chen C, Procko E, Pant S, Lam K, Patel J, Choi R, Hong M, Joshi D, Bolton E, Tajkhorshid E, Chung HJ. Sci Rep. 2020 Mar 16;10(1):4756. doi: 10.1038/s41598-020-61697-6. PubMed
Caldendrin and myosin V regulate synaptic spine apparatus localization via ER stabilization in dendritic spines. Konietzny A, Grendel J, Kadek A, Bucher M, Han Y, Hertrich N, Dekkers DHW, Demmers JAA, Grunewald K, Uetrecht C, Mikhaylova M. EMBO J. 2022 Feb 15;41(4):e106523. doi: 10.15252/embj.2020106523. Epub 2021 Dec 22. PubMed

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