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p1322 HPV-16 E6 Citations (4)

Originally described in: The E6 and E7 genes of the human papillomavirus type 16 together are necessary and sufficient for transformation of primary human keratinocytes.
Münger K, Phelps WC, Bubb V, Howley PM, Schlegel R J Virol. 1989 Oct . 63(10):4417-21.

Articles Citing p1322 HPV-16 E6

Human papillomavirus 16 load and E2/E6 ratio in HPV16-positive women: biomarkers for cervical intraepithelial neoplasia >or=2 in a liquid-based cytology setting? Boulet GA, Benoy IH, Depuydt CE, Horvath CA, Aerts M, Hens N, Vereecken AJ, Bogers JJ. Cancer Epidemiol Biomarkers Prev. 2009 Nov;18(11):2992-9. doi: 10.1158/1055-9965.EPI-09-0025. Epub 2009 Oct 27. PubMed
Rb-dependent cellular senescence, multinucleation and susceptibility to oncogenic transformation through PKC scaffolding by SSeCKS/AKAP12. Akakura S, Nochajski P, Gao L, Sotomayor P, Matsui S, Gelman IH. Cell Cycle. 2010 Dec 1;9(23):4656-65. Epub 2010 Dec 1. PubMed
The Human Papillomavirus E6 Oncoprotein Targets USP15 and TRIM25 To Suppress RIG-I-Mediated Innate Immune Signaling. Chiang C, Pauli EK, Biryukov J, Feister KF, Meng M, White EA, Munger K, Howley PM, Meyers C, Gack MU. J Virol. 2018 Feb 26;92(6). pii: JVI.01737-17. doi: 10.1128/JVI.01737-17. Print 2018 Mar 15. PubMed
Acute HSF1 depletion induces cellular senescence through the MDM2-p53-p21 pathway in human diploid fibroblasts. Oda T, Sekimoto T, Kurashima K, Fujimoto M, Nakai A, Yamashita T. J Cell Sci. 2018 May 8;131(9). pii: jcs.210724. doi: 10.1242/jcs.210724. PubMed

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