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EML4-ALK induces cellular senescence in mortal normal human cells and promotes anchorage-independent growth in hTERT-transduced normal human cells.

Miyanaga A, Matsumoto M, Beck JA, Horikawa I, Oike T, Okayama H, Tanaka H, Burkett SS, Robles AI, Khan M, Lissa D, Seike M, Gemma A, Mano H, Harris CC
BMC Cancer. 2021 Mar 24;21(1):310. doi: 10.1186/s12885-021-07905-6. (Link opens in a new window) PubMed (Link opens in a new window) Article

Plasmids from Article

ID Plasmid Purpose
259519pLenti6.3-EML4-ALKExpresses a fusion oncogene (EML4-ALK) in mammalian cells

Antibodies from Article