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IF-GFP-ATRX Citations (6)

Originally described in: The ATRX cDNA is prone to bacterial IS10 element insertions that alter its structure.
Valle-Garcia D, Griffiths LM, Dyer MA, Bernstein E, Recillas-Targa F Springerplus. 2014 May 2;3:222. doi: 10.1186/2193-1801-3-222. eCollection 2014.
PubMed Journal

Articles Citing IF-GFP-ATRX

Articles
The ATRX cDNA is prone to bacterial IS10 element insertions that alter its structure. Valle-Garcia D, Griffiths LM, Dyer MA, Bernstein E, Recillas-Targa F. Springerplus. 2014 May 2;3:222. doi: 10.1186/2193-1801-3-222. eCollection 2014. PubMed

Associated Plasmids

Genetic Inactivation of ATRX Leads to a Decrease in the Amount of Telomeric Cohesin and Level of Telomere Transcription in Human Glioma Cells. Eid R, Demattei MV, Episkopou H, Auge-Gouillou C, Decottignies A, Grandin N, Charbonneau M. Mol Cell Biol. 2015 Aug;35(16):2818-30. doi: 10.1128/MCB.01317-14. Epub 2015 Jun 8. PubMed
Atrx inactivation drives disease-defining phenotypes in glioma cells of origin through global epigenomic remodeling. Danussi C, Bose P, Parthasarathy PT, Silberman PC, Van Arnam JS, Vitucci M, Tang OY, Heguy A, Wang Y, Chan TA, Riggins GJ, Sulman EP, Lang F, Creighton CJ, Deneen B, Miller CR, Picketts DJ, Kannan K, Huse JT. Nat Commun. 2018 Mar 13;9(1):1057. doi: 10.1038/s41467-018-03476-6. PubMed
Non-invasive assessment of telomere maintenance mechanisms in brain tumors. Viswanath P, Batsios G, Mukherjee J, Gillespie AM, Larson PEZ, Luchman HA, Phillips JJ, Costello JF, Pieper RO, Ronen SM. Nat Commun. 2021 Jan 4;12(1):92. doi: 10.1038/s41467-020-20312-y. PubMed
ATRX and RECQ5 define distinct homologous recombination subpathways. Elbakry A, Juhasz S, Chan KC, Lobrich M. Proc Natl Acad Sci U S A. 2021 Jan 19;118(3). pii: 2010370118. doi: 10.1073/pnas.2010370118. PubMed
Two opposing gene expression patterns within ATRX aberrant neuroblastoma. van Gerven MR, Schild L, van Arkel J, Koopmans B, Broeils LA, Meijs LAM, van Oosterhout R, van Noesel MM, Koster J, van Hooff SR, Molenaar JJ, van den Boogaard ML. PLoS One. 2023 Aug 4;18(8):e0289084. doi: 10.1371/journal.pone.0289084. eCollection 2023. PubMed

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