AAV-mOXT-hM3Dq-mCherry-WPRE Citations (4)

Originally described in: Exogenous and evoked oxytocin restores social behavior in the Cntnap2 mouse model of autism.
Penagarikano O, Lazaro MT, Lu XH, Gordon A, Dong H, Lam HA, Peles E, Maidment NT, Murphy NP, Yang XW, Golshani P, Geschwind DH Sci Transl Med. 2015 Jan 21;7(271):271ra8. doi: 10.1126/scitranslmed.3010257. PubMed Journal

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Articles Citing AAV-mOXT-hM3Dq-mCherry-WPRE

Articles
Oxytocin normalizes altered circuit connectivity for social rescue of the Cntnap2 knockout mouse. Choe KY, Bethlehem RAI, Safrin M, Dong H, Salman E, Li Y, Grinevich V, Golshani P, DeNardo LA, Penagarikano O, Harris NG, Geschwind DH. Neuron. 2022 Mar 2;110(5):795-808.e6. doi: 10.1016/j.neuron.2021.11.031. Epub 2021 Dec 20. PubMed
A dorsal CA2 to ventral CA1 circuit contributes to oxytocinergic modulation of long-term social recognition memory. Tsai TC, Fang YS, Hung YC, Hung LC, Hsu KS. J Biomed Sci. 2022 Jul 10;29(1):50. doi: 10.1186/s12929-022-00834-x. PubMed
Optimization of AAV vectors for transactivator-regulated enhanced gene expression within targeted neuronal populations. Kojima L, Seiriki K, Rokujo H, Nakazawa T, Kasai A, Hashimoto H. iScience. 2024 May 3;27(6):109878. doi: 10.1016/j.isci.2024.109878. eCollection 2024 Jun 21. PubMed
Functional disruption of oxytocin projections participates atypical social and anxiety-like behaviours in BTBR mouse model of autism. Higuchi Y, Ozawa A, Kobayashi R, Konno T, Arakawa H. Open Biol. 2025 Aug;15(8):240387. doi: 10.1098/rsob.240387. Epub 2025 Aug 27. PubMed

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