LentiCRISPRv2-ATG5 Citations (7)
Originally described in: TRIM5alpha degradation via autophagy is not required for retroviral restriction.Imam S, Talley S, Nelson RS, Dharan A, O'Connor C, Hope TJ, Campbell EM J Virol. 2016 Jan 13. pii: JVI.03033-15. PubMed Journal
Articles Citing LentiCRISPRv2-ATG5
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| Measurement of autophagic flux in humans: an optimized method for blood samples. Bensalem J, Hattersley KJ, Hein LK, Teong XT, Carosi JM, Hassiotis S, Grose RH, Fourrier C, Heilbronn LK, Sargeant TJ. Autophagy. 2021 Oct;17(10):3238-3255. doi: 10.1080/15548627.2020.1846302. Epub 2020 Dec 11. PubMed |
| TRIB2 modulates proteasome function to reduce ubiquitin stability and protect liver cancer cells against oxidative stress. Guo S, Chen Y, Yang Y, Zhang X, Ma L, Xue X, Qiao Y, Wang J. Cell Death Dis. 2021 Jan 7;12(1):42. doi: 10.1038/s41419-020-03299-8. PubMed |
| LC3B is lipidated to large lipid droplets during prolonged starvation for noncanonical autophagy. Omrane M, Ben M'Barek K, Santinho A, Nguyen N, Nag S, Melia TJ, Thiam AR. Dev Cell. 2023 Jul 24;58(14):1266-1281.e7. doi: 10.1016/j.devcel.2023.05.009. Epub 2023 Jun 13. PubMed |
| Mutations accumulated in the Spike of SARS-CoV-2 Omicron allow for more efficient counteraction of the restriction factor BST2/Tetherin. Shi Y, Simpson S, Chen Y, Aull H, Benjamin J, Serra-Moreno R. PLoS Pathog. 2024 Jan 8;20(1):e1011912. doi: 10.1371/journal.ppat.1011912. eCollection 2024 Jan. PubMed |
| RAB5 is a host dependency factor for the generation of SARS-CoV-2 replication organelles. Chen Y, Klute S, Sparrer KMJ, Serra-Moreno R. mBio. 2025 May 14;16(5):e0331424. doi: 10.1128/mbio.03314-24. Epub 2025 Apr 1. PubMed |
| Rapid hepatitis C virus replication machinery removal after antiviral treatment with DAA monitored by multimodal imaging. Castro V, Calvo G, Perez-Berna AJ, Mamprin K, Kapishnikov S, Rogers D, O'Connor S, Sheridan P, Fahy K, Pereiro E, Gastaminza P. Structure. 2025 Sep 4;33(9):1547-1564.e4. doi: 10.1016/j.str.2025.05.015. Epub 2025 Jun 18. PubMed |
| Liver CYP4A autophagic lysosomal degradation: A major role for the autophagic receptor SQSTM1/p62 through an uncommon target interaction site. He L, Kwon D, Trnka MJ, Liu Y, Yang J, Li KH, Totah RA, Johnson EF, Burlingame AL, Correia MA. Mol Pharmacol. 2025 Sep;107(9):100061. doi: 10.1016/j.molpha.2025.100061. Epub 2025 Jul 11. PubMed |
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