Overcoming the Silencing of Doxycycline-Inducible Promoters in hiPSC-derived Cardiomyocytes.
Guichardaz M, Bottini S, Balmas E, Bertero A
Open Res Eur. 2024 Dec 18;4:266. doi: 10.12688/openreseurope.19024.1. eCollection 2024.
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Plasmids from Article
| ID | Plasmid | Purpose |
|---|---|---|
| 230050 | pAAV-puro_TRE3VG_EGFP-Responder | It presents the TRE3VG inducible promoter allowing the dox-dependent inducible activation of EGFP through the OPTi-OX platform |
| 230051 | pAAV-puro_T11_EGFP-Responder | It presents the T11 inducible promoter allowing the dox-dependent inducible activation of EGFP through the OPTi-OX platform |
| 230052 | pAAV-puro_TRE3VG_mCherry-Responder | It presents the TRE3VG inducible promoter allowing the dox-dependent inducible activation of mCherry through the OPTi-OX platform |
| 230053 | pAAV-puro_UCOE_TRE3VG_mCherry-Responder | It presents the UCOE sequence and the TRE3VG inducible promoter allowing a more stable dox-dependent inducible activation of mCherry though the OPTi-OX platform across differentiation of hiPSCs |
| 230054 | pCLYBL-Neo_TRE3VG_mCherry_Responder | It presents the TRE3VG inducible promoter allowing the dox-dependent inducible activation of mCherry through the OPTi-OX platform. The CLYBL GSH supports higher transgene expression |